- Secondhand smoke exposure in childhood is linked to increased risk for adult Afib.
- Some of these childhood-exposed adults also smoke, which may be a factor, but analyses were adjusted for family clusters and common Afib risk factors.
Why this matters
- Editorial: these findings should remind clinicians to ask about tobacco use, emphasise to parents the need to avoid exposing their children, and offer cessation interventions where warranted at every visit.
- 14.3% of offspring cohort developed Afib during the median 40.5 (interquartile range, 33.3-41.9) years of follow-up.
- Overall incidence: 4.02 per 1000 person-years.
- With each pack-day increase in parental smoking, Afib incidence among exposed children is increased by 18% (adjusted HR, 1.18; P=.04).
- These children also were more likely themselves to smoke (aOR, 1.34; P<.001 which contributed in part ci to the afib risk.>
- Smoking in the offspring cohort raised Afib risk by 32% (adjusted HR, 1.32; P=.002).
- Study of data from Framingham Offspring cohort (n=2816) and their parents in the original Framingham Heart Study cohort.
- Funding: University of California; American Heart Association; NIH; others.
- For 45% of the offspring cohort, parental smoking status was not available, so their data were not included.