- Obesity is a key metabolic factor linked to urinary incontinence (UI) in both men and women, and to overactive bladder syndrome (OAB) in women.
Why this matters
- "Recent studies suggest that metabolic factors may play a role in the development of [OAB], [UI], and other lower urinary tract symptoms (LUTS)," say researchers.
- Researchers studied adult patients seeking LUTS treatment (N=920), assessing urinary symptoms and metabolic factors.
- Funding: NIH.
- Central obesity (per each 10-cm increment in waist size) was associated with (ORs):
- UI in both sexes: 1.16 (P=.008);
- stress UI (SUI) in women: 1.27 (P=.008);
- urgent UI (UUI) in both sexes: 1.24 (P=0.001); and
- OAB in women: 1.248 (P=.003).
- General obesity (per each 5-unit increase in BMI) was associated with:
- UI in both sexes: 1.443 (P=.028);
- SUI in women: 1.931 (P=.023);
- UUI in both sexes: 1.857 (P=.001);
- OAB in women: 2.432 (P<.001>
- Dyslipidemia was associated with nocturia ≥2: OR, 1.547 (P=.010).
- Researchers did not measure fasting glucose, HbA1c, high-density lipoprotein cholesterol, or triglyceride levels to fully assess metabolic syndrome.